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The Nucleosome Remodelling and Deacetylation complex restricts Mediator access to enhancers to control transcription
Maria Xenophontos, Nicola Reynolds, Sarah Gharbi, Ewan Johnstone, Jason Signolet, Robin Floyd, Meryem Ralser, Susanne Bornelöv, Sabine Dietmann, Remco Loos, Paul Bertone, View ORCID ProfileBrian Hendrich
doi: https://doi.org/10.1101/103192
Maria Xenophontos
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
2European Bioinformatics Institute, European Molecular Biology Laboratory (EMBL), Wellcome Trust Genome Campus, Cambridge CB10 1SD, United Kingdom
Nicola Reynolds
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
Sarah Gharbi
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
Ewan Johnstone
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
2European Bioinformatics Institute, European Molecular Biology Laboratory (EMBL), Wellcome Trust Genome Campus, Cambridge CB10 1SD, United Kingdom
Jason Signolet
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
3Department of Biochemistry, University of Cambridge, Tennis Court Road, Cambridge CB2 1QR United Kingdom
Robin Floyd
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
Meryem Ralser
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
Susanne Bornelöv
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
Sabine Dietmann
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
2European Bioinformatics Institute, European Molecular Biology Laboratory (EMBL), Wellcome Trust Genome Campus, Cambridge CB10 1SD, United Kingdom
Remco Loos
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
2European Bioinformatics Institute, European Molecular Biology Laboratory (EMBL), Wellcome Trust Genome Campus, Cambridge CB10 1SD, United Kingdom
Paul Bertone
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
2European Bioinformatics Institute, European Molecular Biology Laboratory (EMBL), Wellcome Trust Genome Campus, Cambridge CB10 1SD, United Kingdom
Brian Hendrich
1Wellcome Trust – Medical Research Council Stem Cell Institute, University of Cambridge, Gleeson Building, Tennis Court Road, Cambridge CB2 1QR United Kingdom
3Department of Biochemistry, University of Cambridge, Tennis Court Road, Cambridge CB2 1QR United Kingdom
Article usage
Posted January 25, 2017.
The Nucleosome Remodelling and Deacetylation complex restricts Mediator access to enhancers to control transcription
Maria Xenophontos, Nicola Reynolds, Sarah Gharbi, Ewan Johnstone, Jason Signolet, Robin Floyd, Meryem Ralser, Susanne Bornelöv, Sabine Dietmann, Remco Loos, Paul Bertone, Brian Hendrich
bioRxiv 103192; doi: https://doi.org/10.1101/103192
The Nucleosome Remodelling and Deacetylation complex restricts Mediator access to enhancers to control transcription
Maria Xenophontos, Nicola Reynolds, Sarah Gharbi, Ewan Johnstone, Jason Signolet, Robin Floyd, Meryem Ralser, Susanne Bornelöv, Sabine Dietmann, Remco Loos, Paul Bertone, Brian Hendrich
bioRxiv 103192; doi: https://doi.org/10.1101/103192
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