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- September 15, 2015.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC 4.0 International license.
Author Information
- Athma A. Paia12,
- Golshid Baharianb,c1,
- Ariane Page Sabourinc,
- Yohann Nédélecb,c,
- Jean-Christophe Grenierc,
- Katherine J. Siddled,
- Anne Dumainec,
- Vania Yotovac,
- Christopher B. Burgea,e and
- Luis B. Barreiroc,f,2
- aDepartment of Biology, Massachusetts Institute of Technology, Cambridge, MA, USA
- bDepartment of Biochemistry, Faculty of Medicine, University of Montreal, Montreal, QC, Canada
- cSainte-Justine Hospital Research Centre, Montreal, QC, Canada
- dDepartment of Organismic and Evolutionary Biology, FAS Center for Systems Biology, Harvard University, Cambridge, MA, USA
- eDepartment of Biological Engineering, Massachusetts Institute of Technology, Cambridge, MA, USA
- fDepartment of Pediatrics, Faculty of Medicine, University of Montreal, Montreal, QC, Canada
- ↵2To whom correspondence should be addressed. E-mail: athma{at}mit.edu and luis.barreiro{at}umontreal.ca
↵1 These authors contributed equally to this work.
Posted September 15, 2015.
Widespread shortening of 3’ untranslated regions and increased exon inclusion characterize the human macrophage response to infection
