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To what extent are the terminal stages of sepsis, septic shock, SIRS, and multiple organ dysfunction syndrome actually driven by a prion/amyloid form of fibrin?
Douglas B. Kell, Etheresia Pretorius
doi: https://doi.org/10.1101/057851
Douglas B. Kell
1School of Chemistry, The University of Manchester, 131, Princess St, MANCHESTER M1 7DN, Lancs, UK
2The Manchester Institute of Biotechnology, The University of Manchester, 131, Princess St, MANCHESTER M1 7DN, Lancs, UK
3Centre for Synthetic Biology of Fine and Speciality Chemicals, The University of Manchester, 131, Princess St, MANCHESTER M1 7DN, Lancs, UK
Etheresia Pretorius
4Department of Physiology, Faculty of Health Sciences, University of Pretoria, Arcadia 0007, South Africa.

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Posted June 10, 2016.
To what extent are the terminal stages of sepsis, septic shock, SIRS, and multiple organ dysfunction syndrome actually driven by a prion/amyloid form of fibrin?
Douglas B. Kell, Etheresia Pretorius
bioRxiv 057851; doi: https://doi.org/10.1101/057851
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