Article Information
History
- January 18, 2017.
Article Versions
- Version 1 (June 29, 2016 - 14:25).
- You are viewing Version 2, the most recent version of this article.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Adam Liska, MSc1,2,
- Alice Bertero, PhD1,3,
- Ryszard Gomolka, MSc1,
- Mara Sabbioni, MSc4,
- Alberto Galbusera, MSc1,
- Noemi Barsotti, MSc3,
- Stefano Panzeri, PhD5,
- Maria Luisa Scattoni, PhD4,
- Massimo Pasqualetti, PhD1,3 and
- Alessandro Gozzi, PhD1,*
- 1Functional Neuroimaging Laboratory, Center for Neuroscience and Cognitive Systems @ UniTn, Istituto Italiano di Tecnologia, Rovereto, Italy
- 2CIMeC, Center for Mind/Brain Sciences, University of Trento, Rovereto, Italy
- 3Department of Biology, Unit of Cell and Developmental Biology, University of Pisa, Pisa, Italy
- 4Istituto Superiore di Sanità, Neurotoxicology and Neuroendocrinology Section, Department of Cell Biology and Neurosciences, Rome, Italy
- 5Neural Computation Laboratory, Center for Neuroscience and Cognitive Systems @ UniTn, Istituto Italiano di Tecnologia, Rovereto, Italy
- ↵*Corresponding author:
Alessandro Gozzi, Functional Neuroimaging Laboratory, Center for Neuroscience and Cognitive Systems @ UniTn, Istituto Italiano di Tecnologia, 38068, Rovereto, Italy Tel: +39 335 353 830 alessandro.gozzi{at}iit.it
Posted January 18, 2017.
Homozygous loss of autism-risk gene CNTNAP2 results in reduced local and long-range prefrontal functional connectivity
