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A systematic comparison reveals substantial differences in chromosomal versus episomal encoding of enhancer activity
Fumitaka Inoue, Martin Kircher, Beth Martin, Gregory M. Cooper, Daniela M. Witten, Michael T. McManus, Nadav Ahituv, Jay Shendure
doi: https://doi.org/10.1101/061606
Fumitaka Inoue
1Department of Bioengineering and Therapeutic Sciences, Institute for Human Genetics, University of California San Francisco, San Francisco CA, USA
Martin Kircher
2Department of Genome Sciences, University of Washington, Seattle WA, USA
Beth Martin
2Department of Genome Sciences, University of Washington, Seattle WA, USA
Gregory M. Cooper
3HudsonAlpha Institute for Biotechnology, Huntsville AL, USA
Daniela M. Witten
4Departments of Statistics and Biostatistics, University of Washington, Seattle WA, USA
Michael T. McManus
5Department of Microbiology and Immunology, UCSF Diabetes Center, Keck Center for Noncoding RNA, University of California, San Francisco, San Francisco, CA, USA
Nadav Ahituv
1Department of Bioengineering and Therapeutic Sciences, Institute for Human Genetics, University of California San Francisco, San Francisco CA, USA
Jay Shendure
2Department of Genome Sciences, University of Washington, Seattle WA, USA
6Howard Hughes Medical Institute, Seattle WA, USA
Article usage
Posted June 30, 2016.
A systematic comparison reveals substantial differences in chromosomal versus episomal encoding of enhancer activity
Fumitaka Inoue, Martin Kircher, Beth Martin, Gregory M. Cooper, Daniela M. Witten, Michael T. McManus, Nadav Ahituv, Jay Shendure
bioRxiv 061606; doi: https://doi.org/10.1101/061606
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