Article Information
History
- July 19, 2016.
Article Versions
- You are currently viewing Version 1 of this article (July 19, 2016 - 13:46).
- View Version 2, the most recent version of this article.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.
Author Information
- Sean G. Byars1,2,*,
- Qinqin Huang1,
- Lesley-Ann Gray1,2,
- Samuli Ripatti3,4,5,
- Gad Abraham1,2,
- Stephen C. Stearns6 and
- Michael Inouye1,2,3,*
- 1Centre for Systems Genomics, School of BioSciences, The University of Melbourne, Parkville 3010, Victoria, Australia
- 2Department of Pathology, The University of Melbourne, Parkville, Victoria 3010, Australia
- 3National Institute for Health and Welfare, Helsinki, Finland
- 4Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, Cambridge, United Kingdom
- 5Department of Public Health, University of Helsinki, Helsinki, Finland
- 6Department of Ecology and Evolutionary Biology, Yale University, New Haven, CT, USA
- ↵* Corresponding authors: Sean Byars (sean.byars{at}unimelb.edu.au), Michael Inouye (minouye{at}unimelb.edu.au)
Posted July 19, 2016.
Genetic loci associated with coronary artery disease harbor evidence of selection and antagonistic pleiotropy
