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Transcriptional repression by ApiAP2 factors is central to chronic toxoplasmosis
Joshua B. Radke, Danielle Worth, Dong-Pyo Hong, Sherri Huang, William J. Sullivan Jr, Emma H. Wilson, Michael W. White
doi: https://doi.org/10.1101/100628
Joshua B. Radke
1Department of Global Health and Florida Center for Drug Discovery and Innovation, University of South Florida, Tampa, FL 33612, USA
Danielle Worth
2Division of Biomedical Sciences, School of Medicine, University of California, Riverside, Riverside, California, USA
Dong-Pyo Hong
1Department of Global Health and Florida Center for Drug Discovery and Innovation, University of South Florida, Tampa, FL 33612, USA
Sherri Huang
3Department of Pharmacology and Toxicology, Indiana University School of Medicine, Indianapolis, IN 46202, USA
William J. Sullivan Jr
3Department of Pharmacology and Toxicology, Indiana University School of Medicine, Indianapolis, IN 46202, USA
Emma H. Wilson
2Division of Biomedical Sciences, School of Medicine, University of California, Riverside, Riverside, California, USA
Michael W. White
1Department of Global Health and Florida Center for Drug Discovery and Innovation, University of South Florida, Tampa, FL 33612, USA
Posted January 22, 2017.
Transcriptional repression by ApiAP2 factors is central to chronic toxoplasmosis
Joshua B. Radke, Danielle Worth, Dong-Pyo Hong, Sherri Huang, William J. Sullivan Jr, Emma H. Wilson, Michael W. White
bioRxiv 100628; doi: https://doi.org/10.1101/100628
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