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Fragile dynamics enable diverse genomic determinants to influence arrhythmia propensity
Tim J Kamerzell, Eric A Sobie, Kai-Chen Yang, Jeanne M Nerbonne, Calum A MacRae, Ravi Iyengar
doi: https://doi.org/10.1101/101162
Tim J Kamerzell
1Department of Pharmacology and Systems Therapeutics and Systems Biology Center New York, Icahn School of Medicine at Mount Sinai, New York City, NY, USA.
2Department of Developmental Biology and Center for Cardiovascular Research, Division of Cardiology, Department of Internal Medicine, Washington University School of Medicine, Saint Louis, MO, USA.
3Brigham and Women's Hospital, Cardiovascular Division, Boston, MA, USA and Harvard Medical School, Boston, MA, USA.
Eric A Sobie
1Department of Pharmacology and Systems Therapeutics and Systems Biology Center New York, Icahn School of Medicine at Mount Sinai, New York City, NY, USA.
Kai-Chen Yang
2Department of Developmental Biology and Center for Cardiovascular Research, Division of Cardiology, Department of Internal Medicine, Washington University School of Medicine, Saint Louis, MO, USA.
Jeanne M Nerbonne
2Department of Developmental Biology and Center for Cardiovascular Research, Division of Cardiology, Department of Internal Medicine, Washington University School of Medicine, Saint Louis, MO, USA.
Calum A MacRae
3Brigham and Women's Hospital, Cardiovascular Division, Boston, MA, USA and Harvard Medical School, Boston, MA, USA.
Ravi Iyengar
1Department of Pharmacology and Systems Therapeutics and Systems Biology Center New York, Icahn School of Medicine at Mount Sinai, New York City, NY, USA.

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Posted January 18, 2017.
Fragile dynamics enable diverse genomic determinants to influence arrhythmia propensity
Tim J Kamerzell, Eric A Sobie, Kai-Chen Yang, Jeanne M Nerbonne, Calum A MacRae, Ravi Iyengar
bioRxiv 101162; doi: https://doi.org/10.1101/101162
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