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- August 1, 2017.
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- Version 1 (May 23, 2017 - 10:42).
- You are viewing Version 2, the most recent version of this article.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.
Author Information
- Stefano Suzzi1,
- Reiner Ahrendt1,
- Stefan Hans1,
- Svetlana A. Semenova2,
- Saygın Bilican1,
- Shady Sayed1,
- Sylke Winkler3,
- Sandra Spieß1,
- Jan Kaslin1#a,
- Pertti Panula2 and
- Michael Brand1,*
- 1 Biotechnology Center (BIOTEC) and Center for Regenerative Therapies Technische Universität Dresden (CRTD), Dresden, Germany;
- 2 Neuroscience Center, Institute of Biomedicine/Anatomy, University of Helsinki, Helsinki, Finland;
- 3 Max Planck Institute of Molecular Cell Biology and Genetics (MPI-CBG), Dresden, Germany;
- ↵* Corresponding author, e-mail: michael.brand{at}tu-dresden.de
Posted August 01, 2017.
Loss of lrrk2 impairs dopamine catabolism, cell proliferation, and neuronal regeneration in the zebrafish brain
