RANKL signaling sustains primary tumor growth in genetically engineered mouse models of lung adenocarcinoma

Julien Faget; Caroline Contat; Nadine Zangger; Solange Peters; Etienne Meylan

doi:10.1101/142620

Article Information

doi

https://doi.org/10.1101/142620

History

May 26, 2017.

The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY 4.0 International license.

Author Information

Julien Faget1,
Caroline Contat1,
Nadine Zangger1,2,
Solange Peters3 and
Etienne Meylan1,*

¹Swiss Institute for Experimental Cancer Research, School of Life Sciences, Ecole Polytechnique Fédérale de Lausanne, CH-1015 Lausanne, Switzerland.
²Bioinformatics Core Facility, Swiss Institute of Bioinformatics, CH-1015 Lausanne, Switzerland.
³Department of Oncology, Centre Hospitalier Universitaire Vaudois, University of Lausanne, CH-1011 Lausanne, Switzerland.

↵*Corresponding author:
Etienne Meylan, Swiss Institute for Experimental Cancer Research, School of Life Sciences, Ecole Polytechnique Fédérale de Lausanne, Station 19, CH-1015 Lausanne, Switzerland, Phone: +41 (0) 21 693 7247, Fax: +41 (0) 21 693 7210, e-mail: etienne.meylan{at}epfl.ch