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Remodeling of ER-exit sites initiates a membrane supply pathway for autophagosome biogenesis
Liang Ge, Min Zhang, Samuel J Kenny, Dawei Liu, Miharu Maeda, Kota Saito, Anandita Mathur, Ke Xu, View ORCID ProfileRandy Schekman
doi: https://doi.org/10.1101/168518
Liang Ge
1Howard Hughes Medical Institute, and Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720;
Min Zhang
1Howard Hughes Medical Institute, and Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720;
Samuel J Kenny
2Department of Chemistry, University of California, Berkeley, CA 94720
Dawei Liu
1Howard Hughes Medical Institute, and Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720;
Miharu Maeda
3Department of Physiological Chemistry, Graduate School of Pharmaceutical Sciences, University of Tokyo, Tokyo 113-0033, Japan
Kota Saito
3Department of Physiological Chemistry, Graduate School of Pharmaceutical Sciences, University of Tokyo, Tokyo 113-0033, Japan
Anandita Mathur
1Howard Hughes Medical Institute, and Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720;
Ke Xu
2Department of Chemistry, University of California, Berkeley, CA 94720
Randy Schekman
1Howard Hughes Medical Institute, and Department of Molecular and Cell Biology, University of California, Berkeley, CA 94720;
Posted July 25, 2017.
Remodeling of ER-exit sites initiates a membrane supply pathway for autophagosome biogenesis
Liang Ge, Min Zhang, Samuel J Kenny, Dawei Liu, Miharu Maeda, Kota Saito, Anandita Mathur, Ke Xu, Randy Schekman
bioRxiv 168518; doi: https://doi.org/10.1101/168518
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