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The conserved herpesviral kinase ORF36 activates B2 retrotransposons during murine gammaherpesvirus infection

View ORCID ProfileAaron M. Schaller, View ORCID ProfileJessica Tucker, Ian Willis, View ORCID ProfileBritt A. Glaunsinger
doi: https://doi.org/10.1101/2020.01.06.895789
Aaron M. Schaller
1Department of Molecular and Cell Biology, University of California Berkeley, Berkeley, CA, United States of America
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  • ORCID record for Aaron M. Schaller
Jessica Tucker
2Department of Plant & Microbial Biology, University of California Berkeley, Berkeley, CA, United States of America
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Ian Willis
3Departments of Biochemistry and Systems and Computational Biology, Albert Einstein College of Medicine, Bronx, NY, United States of America
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Britt A. Glaunsinger
1Department of Molecular and Cell Biology, University of California Berkeley, Berkeley, CA, United States of America
2Department of Plant & Microbial Biology, University of California Berkeley, Berkeley, CA, United States of America
4Howard Hughes Medical Institute, Berkeley, CA, United States of America
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  • For correspondence: glaunsinger@berkeley.edu
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Posted January 06, 2020.
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The conserved herpesviral kinase ORF36 activates B2 retrotransposons during murine gammaherpesvirus infection
Aaron M. Schaller, Jessica Tucker, Ian Willis, Britt A. Glaunsinger
bioRxiv 2020.01.06.895789; doi: https://doi.org/10.1101/2020.01.06.895789
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The conserved herpesviral kinase ORF36 activates B2 retrotransposons during murine gammaherpesvirus infection
Aaron M. Schaller, Jessica Tucker, Ian Willis, Britt A. Glaunsinger
bioRxiv 2020.01.06.895789; doi: https://doi.org/10.1101/2020.01.06.895789

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