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Loss of Caveolin-1 and caveolae leads to increased cardiac cell stiffness and functional decline of the adult zebrafish heart
Dimitrios Grivas, Álvaro González-Rajal, Carlos Guerrero Rodríguez, Ricardo Garcia, View ORCID ProfileJosé Luis de la Pompa
doi: https://doi.org/10.1101/2020.01.16.909267
Dimitrios Grivas
1Intercellular Signalling in Cardiovascular Development and Disease Laboratory, Centro Nacional de Investigaciones Cardiovasculares Carlos III (CNIC), Melchor Fernández Almagro 3, Madrid 28029, Spain
2Ciber de Enfermedades Cardiovasculares, 28029 Madrid, Spain
Álvaro González-Rajal
1Intercellular Signalling in Cardiovascular Development and Disease Laboratory, Centro Nacional de Investigaciones Cardiovasculares Carlos III (CNIC), Melchor Fernández Almagro 3, Madrid 28029, Spain
3Cell Division Lab, ANZAC Research Institute, Gate 3, Hospital Road, Concord 2139, NSW, Australia
Carlos Guerrero Rodríguez
4Materials Science Factory, Instituto de Ciencia de Materiales de Madrid (ICMM), CSIC, 28049 Madrid, Spain
Ricardo Garcia
4Materials Science Factory, Instituto de Ciencia de Materiales de Madrid (ICMM), CSIC, 28049 Madrid, Spain
José Luis de la Pompa
1Intercellular Signalling in Cardiovascular Development and Disease Laboratory, Centro Nacional de Investigaciones Cardiovasculares Carlos III (CNIC), Melchor Fernández Almagro 3, Madrid 28029, Spain
2Ciber de Enfermedades Cardiovasculares, 28029 Madrid, Spain

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Posted January 16, 2020.
Loss of Caveolin-1 and caveolae leads to increased cardiac cell stiffness and functional decline of the adult zebrafish heart
Dimitrios Grivas, Álvaro González-Rajal, Carlos Guerrero Rodríguez, Ricardo Garcia, José Luis de la Pompa
bioRxiv 2020.01.16.909267; doi: https://doi.org/10.1101/2020.01.16.909267
Loss of Caveolin-1 and caveolae leads to increased cardiac cell stiffness and functional decline of the adult zebrafish heart
Dimitrios Grivas, Álvaro González-Rajal, Carlos Guerrero Rodríguez, Ricardo Garcia, José Luis de la Pompa
bioRxiv 2020.01.16.909267; doi: https://doi.org/10.1101/2020.01.16.909267
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