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Passage of transmissible cancers in the Tasmanian devil is due to a dominant, shared peptide motif and a limited repertoire of MHC-I allotypes
A Gastaldello, SH Ramarathinam, A Bailey, R Owen, S Turner, A Kontouli, T Elliott, P Skipp, AW Purcell, HV Siddle
doi: https://doi.org/10.1101/2020.07.03.184416
A Gastaldello
1School of Biological Sciences, University of Southampton, Southampton, UK
SH Ramarathinam
4Department of Biochemistry and Molecular Biology and the Infection and Immunity Program, Biomedicine Discovery Institute, Monash University, Clayton, Victoria, Australia
A Bailey
2Centre for Cancer Immunology, University of Southampton, Southampton, UK
3Institute for Life Sciences, University of Southampton, Southampton, UK
R Owen
1School of Biological Sciences, University of Southampton, Southampton, UK
S Turner
1School of Biological Sciences, University of Southampton, Southampton, UK
A Kontouli
2Centre for Cancer Immunology, University of Southampton, Southampton, UK
T Elliott
2Centre for Cancer Immunology, University of Southampton, Southampton, UK
3Institute for Life Sciences, University of Southampton, Southampton, UK
P Skipp
1School of Biological Sciences, University of Southampton, Southampton, UK
3Institute for Life Sciences, University of Southampton, Southampton, UK
AW Purcell
4Department of Biochemistry and Molecular Biology and the Infection and Immunity Program, Biomedicine Discovery Institute, Monash University, Clayton, Victoria, Australia
HV Siddle
1School of Biological Sciences, University of Southampton, Southampton, UK
3Institute for Life Sciences, University of Southampton, Southampton, UK

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Posted July 04, 2020.
Passage of transmissible cancers in the Tasmanian devil is due to a dominant, shared peptide motif and a limited repertoire of MHC-I allotypes
A Gastaldello, SH Ramarathinam, A Bailey, R Owen, S Turner, A Kontouli, T Elliott, P Skipp, AW Purcell, HV Siddle
bioRxiv 2020.07.03.184416; doi: https://doi.org/10.1101/2020.07.03.184416
Passage of transmissible cancers in the Tasmanian devil is due to a dominant, shared peptide motif and a limited repertoire of MHC-I allotypes
A Gastaldello, SH Ramarathinam, A Bailey, R Owen, S Turner, A Kontouli, T Elliott, P Skipp, AW Purcell, HV Siddle
bioRxiv 2020.07.03.184416; doi: https://doi.org/10.1101/2020.07.03.184416
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