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History
- October 22, 2020.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.
Author Information
- Ayako Y Murayama1,2,
- Ken-ichiro Kuwako1,3,
- Junko Okahara2,4,
- Byoung-Il Bae5,†,
- Misako Okuno2,
- Hiromi Mashiko6,††,
- Tomomi Shimogori6,
- Christopher A Walsh5,7,8,
- Erika Sasaki2,4,# and
- Hideyuki Okano1,2,#
- 1Department of Physiology, Keio University School of Medicine, Tokyo, Japan
- 2Laboratory for Marmoset Neural Architecture, Center for Brain Science, RIKEN, Wako, Japan
- 3Department of Neural and Muscular Physiology, Shimane University School of Medicine, Izumo, Japan
- 4Center Institute of Experimental Animals, Kawasaki, Japan
- 5Division of Genetics and Genomics, Manton Center for Orphan Disease Research, and Howard Hughes Medical Institute, Boston Children’s Hospital, Boston, MA, USA
- 6Laboratory for Molecular Mechanisms of Brain Development, Center for Brain Science, RIKEN, Wako, Japan
- 7Broad Institute of MIT and Harvard, Cambridge, MA, USA
- 8Departments of Pediatrics and Neurology, Harvard Medical School, Boston, MA, USA
- ↵#Correspondence and requests for materials should be addressed to E.S. (email: esasaki{at}ciea.jp) and H.O. (email: hidokano{at}keio.jp)
Posted October 22, 2020.
The polymicrogyria-associated GPR56 promoter preferentially drives gene expression in developing GABAergic neurons in common marmosets
