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Loss of CDC50A function drives Aβ/p3 production via increased β/α-secretase processing of APP
View ORCID ProfileMarc D. Tambini, View ORCID ProfileLuciano D’Adamio
doi: https://doi.org/10.1101/2020.11.12.379636
Marc D. Tambini
Department of Pharmacology, Physiology & Neuroscience New Jersey Medical School, Brain Health Institute, Jacqueline Krieger Klein Center in Alzheimer’s Disease and Neurodegeneration Research, Rutgers, The State University of New Jersey, 185 South Orange Ave, Newark, NJ, 07103, USA
Luciano D’Adamio
Department of Pharmacology, Physiology & Neuroscience New Jersey Medical School, Brain Health Institute, Jacqueline Krieger Klein Center in Alzheimer’s Disease and Neurodegeneration Research, Rutgers, The State University of New Jersey, 185 South Orange Ave, Newark, NJ, 07103, USA
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Posted November 12, 2020.
Loss of CDC50A function drives Aβ/p3 production via increased β/α-secretase processing of APP
Marc D. Tambini, Luciano D’Adamio
bioRxiv 2020.11.12.379636; doi: https://doi.org/10.1101/2020.11.12.379636
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