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The Tec kinase ITK differentially optimizes NFAT, NF-κB, and MAPK signaling during early T cell activation to regulate graded gene induction
View ORCID ProfileMichael P. Gallagher, View ORCID ProfileJames M. Conley, View ORCID ProfilePranitha Vangala, View ORCID ProfileAndrea Reboldi, View ORCID ProfileManuel Garber, View ORCID ProfileLeslie J. Berg
doi: https://doi.org/10.1101/2020.11.12.380725
Michael P. Gallagher
1Department of Pathology, University of Massachusetts Medical School, Worcester, MA 01605
James M. Conley
2Department of Immunology and Microbiology, University of Colorado School of Medicine, Aurora, CO 80045
Pranitha Vangala
1Department of Pathology, University of Massachusetts Medical School, Worcester, MA 01605
Andrea Reboldi
1Department of Pathology, University of Massachusetts Medical School, Worcester, MA 01605
Manuel Garber
1Department of Pathology, University of Massachusetts Medical School, Worcester, MA 01605
Leslie J. Berg
1Department of Pathology, University of Massachusetts Medical School, Worcester, MA 01605
2Department of Immunology and Microbiology, University of Colorado School of Medicine, Aurora, CO 80045
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Posted November 14, 2020.
The Tec kinase ITK differentially optimizes NFAT, NF-κB, and MAPK signaling during early T cell activation to regulate graded gene induction
Michael P. Gallagher, James M. Conley, Pranitha Vangala, Andrea Reboldi, Manuel Garber, Leslie J. Berg
bioRxiv 2020.11.12.380725; doi: https://doi.org/10.1101/2020.11.12.380725
The Tec kinase ITK differentially optimizes NFAT, NF-κB, and MAPK signaling during early T cell activation to regulate graded gene induction
Michael P. Gallagher, James M. Conley, Pranitha Vangala, Andrea Reboldi, Manuel Garber, Leslie J. Berg
bioRxiv 2020.11.12.380725; doi: https://doi.org/10.1101/2020.11.12.380725
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