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KDM2B promotes cell viability by enhancing DNA damage response in canine hemangiosarcoma
Kevin Christian M. Gulay, View ORCID ProfileKeisuke Aoshima, Yuki Shibata, Hironobu Yasui, View ORCID ProfileQin Yan, Atsushi Kobayashi, Takashi Kimura
doi: https://doi.org/10.1101/2020.11.17.387704
Kevin Christian M. Gulay
1Laboratory of Comparative Pathology, Department of Clinical Sciences, Faculty of Veterinary Medicine, Hokkaido University, Sapporo, Hokkaido, 060-0818, Japan
Keisuke Aoshima
1Laboratory of Comparative Pathology, Department of Clinical Sciences, Faculty of Veterinary Medicine, Hokkaido University, Sapporo, Hokkaido, 060-0818, Japan
Yuki Shibata
2Laboratory of Radiation Biology, Department of Applied Veterinary Sciences, Faculty of Veterinary Medicine, Hokkaido University, Sapporo, Hokkaido, 060-0818, Japan
Hironobu Yasui
2Laboratory of Radiation Biology, Department of Applied Veterinary Sciences, Faculty of Veterinary Medicine, Hokkaido University, Sapporo, Hokkaido, 060-0818, Japan
Qin Yan
3Department of Pathology, Yale School of Medicine, New Haven, CT, USA
Atsushi Kobayashi
1Laboratory of Comparative Pathology, Department of Clinical Sciences, Faculty of Veterinary Medicine, Hokkaido University, Sapporo, Hokkaido, 060-0818, Japan
Takashi Kimura
1Laboratory of Comparative Pathology, Department of Clinical Sciences, Faculty of Veterinary Medicine, Hokkaido University, Sapporo, Hokkaido, 060-0818, Japan
Posted November 17, 2020.
KDM2B promotes cell viability by enhancing DNA damage response in canine hemangiosarcoma
Kevin Christian M. Gulay, Keisuke Aoshima, Yuki Shibata, Hironobu Yasui, Qin Yan, Atsushi Kobayashi, Takashi Kimura
bioRxiv 2020.11.17.387704; doi: https://doi.org/10.1101/2020.11.17.387704
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