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Transient genomic instability drives tumorigenesis through accelerated clonal evolution
Ofer Shoshani, Bjorn Bakker, Yin Wang, Dong Hyun Kim, Marcus Maldonado, Matthew A. Demarest, Jon Artates, Ouyang Zhengyu, Adam Mark, Rene Wardenaar, Roman Sasik, Diana C.J. Spierings, Benjamin Vitre, Kathleen Fisch, View ORCID ProfileFloris Foijer, View ORCID ProfileDon W. Cleveland
doi: https://doi.org/10.1101/2020.11.17.387753
Ofer Shoshani
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Bjorn Bakker
2European Research Institute for the Biology of Ageing (ERIBA), University of Groningen, University Medical Center Groningen, 9713 AV, Groningen, The Netherlands
Yin Wang
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Dong Hyun Kim
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Marcus Maldonado
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Matthew A. Demarest
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Jon Artates
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Ouyang Zhengyu
3Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Adam Mark
4Center for Computational Biology & Bioinformatics, Department of Medicine, University of California, San Diego, La Jolla, CA, USA
Rene Wardenaar
2European Research Institute for the Biology of Ageing (ERIBA), University of Groningen, University Medical Center Groningen, 9713 AV, Groningen, The Netherlands
Roman Sasik
4Center for Computational Biology & Bioinformatics, Department of Medicine, University of California, San Diego, La Jolla, CA, USA
Diana C.J. Spierings
2European Research Institute for the Biology of Ageing (ERIBA), University of Groningen, University Medical Center Groningen, 9713 AV, Groningen, The Netherlands
Benjamin Vitre
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
5CRBM, Univ Montpellier, CNRS, Montpellier, France
Kathleen Fisch
4Center for Computational Biology & Bioinformatics, Department of Medicine, University of California, San Diego, La Jolla, CA, USA
Floris Foijer
2European Research Institute for the Biology of Ageing (ERIBA), University of Groningen, University Medical Center Groningen, 9713 AV, Groningen, The Netherlands
Don W. Cleveland
1Ludwig Cancer Research and Department of Cellular and Molecular Medicine, University of California at San Diego, La Jolla, CA, USA
Posted November 18, 2020.
Transient genomic instability drives tumorigenesis through accelerated clonal evolution
Ofer Shoshani, Bjorn Bakker, Yin Wang, Dong Hyun Kim, Marcus Maldonado, Matthew A. Demarest, Jon Artates, Ouyang Zhengyu, Adam Mark, Rene Wardenaar, Roman Sasik, Diana C.J. Spierings, Benjamin Vitre, Kathleen Fisch, Floris Foijer, Don W. Cleveland
bioRxiv 2020.11.17.387753; doi: https://doi.org/10.1101/2020.11.17.387753
Transient genomic instability drives tumorigenesis through accelerated clonal evolution
Ofer Shoshani, Bjorn Bakker, Yin Wang, Dong Hyun Kim, Marcus Maldonado, Matthew A. Demarest, Jon Artates, Ouyang Zhengyu, Adam Mark, Rene Wardenaar, Roman Sasik, Diana C.J. Spierings, Benjamin Vitre, Kathleen Fisch, Floris Foijer, Don W. Cleveland
bioRxiv 2020.11.17.387753; doi: https://doi.org/10.1101/2020.11.17.387753
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