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History
- November 23, 2020.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Görkem Garipler1,7,
- Congyi Lu1,2,7,
- Alexis Morrissey3,
- Lorena S. Lopez-Zepeda4,5,
- Simon E. Vidal6,
- Begüm Aydin1,
- Matthias Stadtfeld6,
- Uwe Ohler4,5,
- Shaun Mahony3,
- Neville E. Sanjana1,2,8 and
- Esteban O. Mazzoni1,8
- 1Department of Biology, New York University, New York, NY 10003, USA
- 2New York Genome Center, New York, NY, 10013 USA
- 3Center for Eukaryotic Gene Regulation, Department of Biochemistry and Molecular Biology, Penn State University, PA 16801, USA
- 4Department of Biology, Humboldt Universität zu Berlin, Berlin 10117, Germany
- 5Berlin Institute for Medical Systems Biology, Max-Delbrück-Center for Molecular Medicine, Berlin 13125, Germany
- 6Skirball Institute of Biomolecular Medicine, Department of Cell Biology, NYU Langone Medical Center, New York, NY 10016, USA
- ↵8Corresponding authors: Neville E. Sanjana (neville{at}sanjanalab.org) & Esteban O. Mazzoni (eom204{at}nyu.edu)
↵7 Equal contribution
Posted November 23, 2020.
The BTB transcription factors ZBTB11 and ZFP131 maintain pluripotency by pausing POL II at pro-differentiation genes
