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- December 21, 2020.
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Author Information
- Elisa Avolio, PhD1,*,
- Monica Gamez, PhD1,
- Kapil Gupta, PhD2,
- Rebecca Foster, PhD1,
- Imre Berger, PhD2,3,
- Massimo Caputo, MD1,
- Andrew Davidson, PhD4,
- Darryl Hill, PhD4 and
- Paolo Madeddu, MD1,*
- 1Bristol Medical School, Translational Health Sciences, University of Bristol, Bristol, United Kingdom
- 2School of Biochemistry, University of Bristol, Bristol, United Kingdom
- 3Max Planck Bristol Centre for Minimal Biology, University of Bristol, Bristol, United Kingdom
- 4School of Cellular and Molecular Medicine, University of Bristol, Bristol, United Kingdom
- ↵*Corresponding authors
Elisa Avolio, PhD, Bristol Medical School, Translational Health Sciences, University of Bristol, Bristol Royal Infirmary, Upper Maudlin Street, BS28HW, Bristol, United Kingdom, Email: elisa.avolio{at}bristol.ac.uk, Paolo Madeddu, MD, Professor of Experimental Cardiovascular Medicine, Bristol Medical School, Translational Health Sciences, University of Bristol, Bristol Royal Infirmary, Upper Maudlin Street, BS28HW, Bristol, United Kingdom, Email: mdprm{at}bristol.ac.uk, Tel and fax: +44 (0) 117 34 23904
Posted December 21, 2020.
The SARS-CoV-2 spike protein disrupts the cooperative function of human cardiac pericytes - endothelial cells through CD147 receptor-mediated signalling: a potential non-infective mechanism of COVID-19 microvascular disease
