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- November 1, 2021.
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- Version 1 (April 8, 2021 - 02:43).
- You are viewing Version 2, the most recent version of this article.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Bailey Lubinski1,
- Maureen H. V. Fernandes2,
- Laura Frazier1,
- Tiffany Tang3,
- Susan Daniel3,
- Diego G. Diel2,
- Javier A. Jaimes1,* and
- Gary R. Whittaker1,4,*
- 1Department of Microbiology & Immunology, College of Veterinary Medicine, Cornell University, Ithaca, NY, 14853, USA
- 2Department of Population Medicine, College of Veterinary Medicine, Cornell University, Ithaca, NY, 14853, USA
- 3Robert Frederick Smith School of Chemical & Biomolecular Engineering, Cornell University, Ithaca, NY, 14853, USA
- 4Master of Public Health Program, Cornell University, Ithaca, NY, 14853, USA
- ↵*Corresponding authors 618 Tower Rd., Ithaca NY 14853, USA, jaj246{at}cornell.edu; grw7{at}cornell.edu
Posted November 01, 2021.
Functional evaluation of the P681H mutation on the proteolytic activation the SARS-CoV-2 variant B.1.1.7 (Alpha) spike
