Article Information
History
- May 3, 2021.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.
Author Information
- Vanessa Byles1,
- Yann Cormerais1,
- Krystle Kalafut1,
- Victor Barrera2,
- James E. Hughes Hallett1,
- Shannan Ho Sui2,
- John M. Asara3,
- Christopher M. Adams4,
- Gerta Hoxhaj1,5,
- Issam Ben-Sahra6 and
- Brendan D. Manning1,7
- 1Department of Molecular Metabolism, Harvard T.H. Chan School of Public Health, Boston, MA, USA
- 2Department of Biostatistics, Harvard T.H. Chan School of Public Health, Boston, MA, USA
- 3Division of Signal Transduction, Beth Israel Deaconess Medical Center and Department of Medicine, Harvard Medical School, Boston, MA, USA
- 4Division of Endocrinology, Metabolism and Nutrition, Mayo Clinic, Rochester, MN, USA
- 5Children’s Medical Center Research Institute, University of Texas Southwestern Medical Center, Dallas, TX, USA
- 6Department of Biochemistry and Molecular Genetics, Northwestern University Feinberg School of Medicine and Robert H. Lurie Comprehensive Cancer Center, Northwestern University, Chicago, IL, USA
- ↵7Correspondence to: bmanning{at}hsph.harvard.edu
Posted May 03, 2021.
Hepatic mTORC1 signaling activates ATF4 as part of its metabolic response to feeding and insulin
