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History
- May 28, 2021.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.
Author Information
- Aaron S. Mendez*,1,
- Michael Ly*,2,
- Angélica M. González-Sánchez2,3,
- Ella Hartenian2,
- Nicholas T. Ingolia2,4,
- Jamie H. Cate2,4,5,6,7 and
- Britt A. Glaunsinger#,1,2,4,5,8
- 1Department of Plant & Microbial Biology, University of California, Berkeley, CA, USA
- 2Department of Molecular and Cell Biology, University of California, Berkeley, CA, USA
- 3Comparative Biochemistry Graduate Program, University of California, Berkeley, CA, USA
- 4California Institute for Quantitative Biosciences, University of California, Berkeley, CA, USA
- 5Innovative Genomics Institute, University of California, Berkeley, CA, USA
- 6Department of Chemistry, University of California, Berkeley, Berkeley, United States
- 7Molecular Biophysics & Integrated Bioimaging Division, Lawrence Berkeley National Laboratory, Berkeley, CA, USA
- 8Howard Hughes Medical Institute, Berkeley, CA, USA
- ↵#corresponding author: glaunsinger{at}berkeley.edu
↵* equal contribution
Posted May 28, 2021.
The N-terminal and central domains of CoV-2 nsp1 play key functional roles in suppression of cellular gene expression and preservation of viral gene expression
