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History
- June 9, 2021.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-ND 4.0 International license.
Author Information
- Maria Papadaki1,
- Theerachat Kampaengsri1,
- Samantha K. Barrick2,
- Stuart G. Campbell3,
- Dirk von Lewinski4,
- Peter P. Rainer4,
- Samantha P. Harris5,
- Michael J. Greenberg2 and
- Jonathan A. Kirk1,†
- 1Department of Cell and Molecular Physiology, Loyola University of Chicago, Maywood, Illinois, USA
- 2Department of Biochemistry and Molecular Biophysics, Washington University in St Louis, St Louis, Missouri, USA
- 3Department of Bioengineering, Yale University, New Haven, Connecticut, USA
- 4Division of Cardiology, Medical University of Graz, Graz, Austria
- 5Department of Cellular and Molecular Medicine, The University of Arizona, Tucson, Arizona, USA
- ↵†Corresponding Author Jonathan A. Kirk, Ph.D., Department of Cell and Molecular Physiology, Loyola University Chicago Stritch School of Medicine, Center for Translational Research, Room 522, 2160 S. First Ave., Maywood, IL 60153, Ph: 708-216-6348, Email: jkirk2{at}luc.edu
Posted June 09, 2021.
Myofilament Glycation in Diabetes Reduces Contractility by Inhibiting Tropomyosin Movement, is Rescued by cMyBPC Domains
