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Increased 20-HETE signaling suppresses neurovascular coupling after ischemic stroke in regions beyond the infarct

View ORCID ProfileZhenzhou Li, View ORCID ProfileHeather L. McConnell, View ORCID ProfileTeresa L. Stackhouse, View ORCID ProfileMartin M. Pike, View ORCID ProfileWenri Zhang, View ORCID ProfileAnusha Mishra
doi: https://doi.org/10.1101/2021.08.25.457547
Zhenzhou Li
1Knight Cardiovascular Institute, Oregon Health & Science University, Portland, OR
2Department Anesthesiology, General Hospital of Ningxia Medical University, Ningxia, China
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Heather L. McConnell
3Department of Neurology, Jungers Center for Neurosciences Research, Oregon Health & Science University, Portland, OR
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Teresa L. Stackhouse
3Department of Neurology, Jungers Center for Neurosciences Research, Oregon Health & Science University, Portland, OR
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Martin M. Pike
4Advanced Imaging Research Center, Oregon Health & Science University, Portland, OR
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Wenri Zhang
5Department of Anesthesiology and Peri-operative Medicine, Oregon Health & Science University, Portland, OR
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Anusha Mishra
1Knight Cardiovascular Institute, Oregon Health & Science University, Portland, OR
3Department of Neurology, Jungers Center for Neurosciences Research, Oregon Health & Science University, Portland, OR
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  • For correspondence: mishraa@ohsu.edu
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Posted August 27, 2021.
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Increased 20-HETE signaling suppresses neurovascular coupling after ischemic stroke in regions beyond the infarct
Zhenzhou Li, Heather L. McConnell, Teresa L. Stackhouse, Martin M. Pike, Wenri Zhang, Anusha Mishra
bioRxiv 2021.08.25.457547; doi: https://doi.org/10.1101/2021.08.25.457547
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Increased 20-HETE signaling suppresses neurovascular coupling after ischemic stroke in regions beyond the infarct
Zhenzhou Li, Heather L. McConnell, Teresa L. Stackhouse, Martin M. Pike, Wenri Zhang, Anusha Mishra
bioRxiv 2021.08.25.457547; doi: https://doi.org/10.1101/2021.08.25.457547

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