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History
- September 8, 2021.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.
Author Information
- Uthman Okalang1,*,
- Bar Mualem Bar-Ner1,*,
- K. Shanmugha Rajan1,*,
- Nehemya Friedman1,
- Saurav Aryal1,
- Katarina Egarmina1,
- Ronen Hope1,
- Netaly Khazanov2,
- Hanoch Senderowitz2,
- Assaf Alon3,
- Deborah Fass3 and
- Shulamit Michaeli1,#
- 1The Mina and Everard Goodman Faculty of Life Sciences and Advanced Materials and Nanotechnology Institute, Bar-Ilan University, Ramat-Gan 52900, Israel.
- 2Chemistry Department, Bar-Ilan University, Ramat-Gan 52900, Israel.
- 3Department of Structural Biology, Weizmann Institute of Science, Rehovot 7610001, Israel
- ↵# Corresponding author: Professor Shulamit Michaeli, The Mina &Everard Goodman Faculty of Life Sciences, Bar-Ilan University, E-mail: Shulamit.Michaeli{at}biu.ac.il
Posted September 08, 2021.
The spliced leader RNA silencing (SLS) pathway in Trypanosoma brucei is induced by perturbations of endoplasmic reticulum, Golgi, or mitochondrial proteins factors and functional analysis of SLS inducing kinase, PK3
