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The proSAAS chaperone provides neuroprotection and attenuates transsynaptic α–synuclein spread in rodent models of Parkinson’s disease
View ORCID ProfileIris Lindberg, Zhan Shu, Hoa Lam, Michael Helwig, Nur Yucer, Alexander Laperle, Clive Svendsen, Donato A. Di Monte, Nigel T. Maidment
doi: https://doi.org/10.1101/2021.09.29.462435
Iris Lindberg
1University of Maryland-Baltimore
Zhan Shu
2University of California-Los Angeles
Hoa Lam
2University of California-Los Angeles
Michael Helwig
3German Center for Neurodegenerative Disease (DZNE)
Nur Yucer
4Cedars-Sinai Medical Center, Los Angeles
Alexander Laperle
4Cedars-Sinai Medical Center, Los Angeles
Clive Svendsen
4Cedars-Sinai Medical Center, Los Angeles
Donato A. Di Monte
3German Center for Neurodegenerative Disease (DZNE)
Nigel T. Maidment
2University of California-Los Angeles

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Posted October 01, 2021.
The proSAAS chaperone provides neuroprotection and attenuates transsynaptic α–synuclein spread in rodent models of Parkinson’s disease
Iris Lindberg, Zhan Shu, Hoa Lam, Michael Helwig, Nur Yucer, Alexander Laperle, Clive Svendsen, Donato A. Di Monte, Nigel T. Maidment
bioRxiv 2021.09.29.462435; doi: https://doi.org/10.1101/2021.09.29.462435
The proSAAS chaperone provides neuroprotection and attenuates transsynaptic α–synuclein spread in rodent models of Parkinson’s disease
Iris Lindberg, Zhan Shu, Hoa Lam, Michael Helwig, Nur Yucer, Alexander Laperle, Clive Svendsen, Donato A. Di Monte, Nigel T. Maidment
bioRxiv 2021.09.29.462435; doi: https://doi.org/10.1101/2021.09.29.462435
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