Article Information
History
- October 14, 2021.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC 4.0 International license.
Author Information
- Haley M. Geertsma1,2,
- Terry R. Suk1,2,
- Konrad M. Ricke1,2,
- Kyra Horsthuis1,2,
- Jean-Louis A. Parmasad1,2,
- Zoe Fisk1,2,
- Steve M. Callaghan1,2 and
- Maxime W.C. Rousseaux1,2,3,4,#
- 1Department of Cellular and Molecular Medicine, University of Ottawa, Ottawa, ON, K1H8M5
- 2University of Ottawa Brain and Mind Research Institute, Ottawa, ON, K1H8M5
- 3Ottawa Institute of Systems Biology, Ottawa, ON, K1H8M5
- 4Eric Poulin Center for Neuromuscular Diseases, Ottawa, ON, K1H8M5
- ↵#Correspondence: max.rousseaux{at}uottawa.ca
Posted October 14, 2021.
Constitutive nuclear accumulation of endogenous alpha-synuclein in mice causes motor dysfunction and cortical atrophy, independent of protein aggregation
