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Mature Neurons’ sensitivity to oxidative stress is epigenetically programmed by alternative splicing and mRNA stability
Yuan Zhou, View ORCID ProfileSherif Rashad, Teiji Tominaga, Kuniyasu Niizuma
doi: https://doi.org/10.1101/2021.12.25.472549
Yuan Zhou
1Department of Neurosurgical Engineering and Translational Neuroscience, Tohoku University Graduate School of Medicine, Sendai, Japan
MDSherif Rashad
1Department of Neurosurgical Engineering and Translational Neuroscience, Tohoku University Graduate School of Medicine, Sendai, Japan
2Department of Neurosurgical Engineering and Translational Neuroscience, Graduate School of Biomedical Engineering, Tohoku University, Sendai, Japan
3Department of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan
MD, PhDTeiji Tominaga
3Department of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan
MD, PhDKuniyasu Niizuma
1Department of Neurosurgical Engineering and Translational Neuroscience, Tohoku University Graduate School of Medicine, Sendai, Japan
2Department of Neurosurgical Engineering and Translational Neuroscience, Graduate School of Biomedical Engineering, Tohoku University, Sendai, Japan
3Department of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan
MD, PhD
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Posted December 25, 2021.
Mature Neurons’ sensitivity to oxidative stress is epigenetically programmed by alternative splicing and mRNA stability
Yuan Zhou, Sherif Rashad, Teiji Tominaga, Kuniyasu Niizuma
bioRxiv 2021.12.25.472549; doi: https://doi.org/10.1101/2021.12.25.472549
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