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Short OGA is targeted to the mitochondria and regulates mitochondrial reactive oxygen species level

Patrick Pagesy, Abdelouhab Bouaboud, Zhihao Feng, Philippe Hulin, Tarik Issad
doi: https://doi.org/10.1101/2021.12.25.474160

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doi 
https://doi.org/10.1101/2021.12.25.474160
History 
  • December 25, 2021.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.

Author Information

  1. Patrick Pagesy1,
  2. Abdelouhab Bouaboud1,
  3. Zhihao Feng1,
  4. Philippe Hulin2 and
  5. Tarik Issad1,*
  1. 1Université de Paris, Institut Cochin, CNRS, INSERM, F-75014 Paris, France
  2. 2Université de Nantes, CHU Nantes, Inserm, CNRS, SFR Santé, Inserm UMS 016, CNRS UMS 3556, F-44000 Nantes, France
  1. ↵*Address correspondence to: Tarik Issad, Institut Cochin, Department of Endocrinology, Metabolism and Diabetes, 24 rue du Faubourg Saint-Jacques, 75014 Paris FRANCE. Tel: + 33 1 44 41 25 67; E-mail: tarik.issad{at}inserm.fr
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Posted December 25, 2021.
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Short OGA is targeted to the mitochondria and regulates mitochondrial reactive oxygen species level
Patrick Pagesy, Abdelouhab Bouaboud, Zhihao Feng, Philippe Hulin, Tarik Issad
bioRxiv 2021.12.25.474160; doi: https://doi.org/10.1101/2021.12.25.474160
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