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Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges

Sourav Roy, Prithwi Ghosh, Abhirup Bandyopadhyay, Sankar Basu
doi: https://doi.org/10.1101/2021.12.29.474439

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doi 
https://doi.org/10.1101/2021.12.29.474439
History 
  • December 29, 2021.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.

Author Information

  1. Sourav Roy1,
  2. Prithwi Ghosh2,
  3. Abhirup Bandyopadhyay3 and
  4. Sankar Basu4,*
  1. 1Dept of Microbiology and Immunology, Brody School of Medicine, East Carolina University, Greenville, NC, 27834, USA
  2. 2Department of Botany, Narajole Raj College, Narajole, Paschim Medinipur, West Bengal, India
  3. 3Theoretical Neurosciences Group, Institute De Neurosciences Des Systems, Aix-Marseille University, France
  4. 4Department of Microbiology, Asutosh College (affiliated to University of Calcutta), 92, Shyama Prasad Mukherjee Rd, Bhowanipore, Kolkata, West Bengal 700026, India
  1. ↵*Corresponding Author’s Email: nemo8130{at}gmail.com
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Posted December 29, 2021.
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Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges
Sourav Roy, Prithwi Ghosh, Abhirup Bandyopadhyay, Sankar Basu
bioRxiv 2021.12.29.474439; doi: https://doi.org/10.1101/2021.12.29.474439
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