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Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges
View ORCID ProfileSourav Roy, View ORCID ProfilePrithwi Ghosh, View ORCID ProfileAbhirup Bandyopadhyay, View ORCID ProfileSankar Basu
doi: https://doi.org/10.1101/2021.12.29.474439
Sourav Roy
1Dept of Microbiology and Immunology, Brody School of Medicine, East Carolina University, Greenville, NC, 27834, USA
Prithwi Ghosh
2Department of Botany, Narajole Raj College, Narajole, Paschim Medinipur, West Bengal, India
Abhirup Bandyopadhyay
3Theoretical Neurosciences Group, Institute De Neurosciences Des Systems, Aix-Marseille University, France
Sankar Basu
4Department of Microbiology, Asutosh College (affiliated to University of Calcutta), 92, Shyama Prasad Mukherjee Rd, Bhowanipore, Kolkata, West Bengal 700026, India
Posted December 29, 2021.
Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges
Sourav Roy, Prithwi Ghosh, Abhirup Bandyopadhyay, Sankar Basu
bioRxiv 2021.12.29.474439; doi: https://doi.org/10.1101/2021.12.29.474439
Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges
Sourav Roy, Prithwi Ghosh, Abhirup Bandyopadhyay, Sankar Basu
bioRxiv 2021.12.29.474439; doi: https://doi.org/10.1101/2021.12.29.474439
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