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Multimodal single cell sequencing of human diabetic kidney disease implicates chromatin accessibility and genetic background in disease progression
View ORCID ProfileParker C. Wilson, View ORCID ProfileYoshiharu Muto, View ORCID ProfileHaojia Wu, Anil Karihaloo, View ORCID ProfileSushrut S. Waikar, View ORCID ProfileBenjamin D. Humphreys
doi: https://doi.org/10.1101/2022.01.28.478204
Parker C. Wilson
1Division of Anatomic and Molecular Pathology, Department of Pathology and Immunology, Washington University in St. Louis, St. Louis, MO, USA
Yoshiharu Muto
2Division of Nephrology, Department of Medicine, Washington University in St. Louis, St. Louis, MO, USA
Haojia Wu
2Division of Nephrology, Department of Medicine, Washington University in St. Louis, St. Louis, MO, USA
Anil Karihaloo
3Novo Nordisk Research Center Seattle Inc, Seattle, WA, USA
Sushrut S. Waikar
4Section of Nephrology, Department of Medicine, Boston University School of Medicine, Boston Medical Center, Boston, MA, USA
Benjamin D. Humphreys
2Division of Nephrology, Department of Medicine, Washington University in St. Louis, St. Louis, MO, USA
5Department of Developmental Biology, Washington University in St. Louis, St. Louis, MO, USA
Posted January 28, 2022.
Multimodal single cell sequencing of human diabetic kidney disease implicates chromatin accessibility and genetic background in disease progression
Parker C. Wilson, Yoshiharu Muto, Haojia Wu, Anil Karihaloo, Sushrut S. Waikar, Benjamin D. Humphreys
bioRxiv 2022.01.28.478204; doi: https://doi.org/10.1101/2022.01.28.478204
Multimodal single cell sequencing of human diabetic kidney disease implicates chromatin accessibility and genetic background in disease progression
Parker C. Wilson, Yoshiharu Muto, Haojia Wu, Anil Karihaloo, Sushrut S. Waikar, Benjamin D. Humphreys
bioRxiv 2022.01.28.478204; doi: https://doi.org/10.1101/2022.01.28.478204
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