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- March 19, 2022.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Ane Korff1,
- Xiaojing Yang1,
- Kevin O’Donovan1,
- Fen Yang1,
- Tushar Patni2,
- Heather Tillman3,
- Abner Gonzalez4,
- Yuh Min Chook4,
- J. Paul Taylor1,5,* and
- Hong Joo Kim1,*
- 1Department of Cell and Molecular Biology, St. Jude Children’s Research Hospital, Memphis, Tennessee, USA
- 2Department of Biostatistics, St. Jude Children’s Research Hospital, Memphis, Tennessee, USA
- 3Department of Pathology, St. Jude Children’s Research Hospital, Memphis, Tennessee, USA
- 4Department of Pharmacology, University of Texas Southwestern Medical Center, Dallas, TX, USA
- 5Howard Hughes Medical Institute, Chevy Chase, Maryland, USA
- ↵*co-senior and co-corresponding authors; email: jpaul.taylor{at}stjude.org
Posted March 19, 2022.
A murine model of hnRNPH2-related neurodevelopmental disorder recapitulates clinical features of human disease and reveals a mechanism for genetic compensation of HNRNPH2
