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Transitional alveolar epithelial cells and microenvironmental stiffness synergistically drive fibroblast activation in three-dimensional hydrogel lung models
Thomas Caracena, Rachel Blomberg, Rukshika S. Hewawasam, David W.H. Riches, View ORCID ProfileChelsea M. Magin
doi: https://doi.org/10.1101/2022.05.24.493246
Thomas Caracena
1Department of Bioengineering, University of Colorado, Denver | Anschutz Medical Campus
Rachel Blomberg
1Department of Bioengineering, University of Colorado, Denver | Anschutz Medical Campus
Rukshika S. Hewawasam
1Department of Bioengineering, University of Colorado, Denver | Anschutz Medical Campus
David W.H. Riches
2Program in Cell Biology, Department of Pediatrics, National Jewish Health
3Division of Pulmonary Sciences and Critical Care Medicine, Department of Medicine, University of Colorado, Anschutz Medical Campus
4Department of Research, Veterans Affairs Eastern Colorado Health Care System
5Department of Immunology and Microbiology, University of Colorado, Anschutz Medical Campus
Chelsea M. Magin
1Department of Bioengineering, University of Colorado, Denver | Anschutz Medical Campus
3Division of Pulmonary Sciences and Critical Care Medicine, Department of Medicine, University of Colorado, Anschutz Medical Campus
6Department of Pediatrics, University of Colorado, Anschutz Medical Campus
Posted May 25, 2022.
Transitional alveolar epithelial cells and microenvironmental stiffness synergistically drive fibroblast activation in three-dimensional hydrogel lung models
Thomas Caracena, Rachel Blomberg, Rukshika S. Hewawasam, David W.H. Riches, Chelsea M. Magin
bioRxiv 2022.05.24.493246; doi: https://doi.org/10.1101/2022.05.24.493246
Transitional alveolar epithelial cells and microenvironmental stiffness synergistically drive fibroblast activation in three-dimensional hydrogel lung models
Thomas Caracena, Rachel Blomberg, Rukshika S. Hewawasam, David W.H. Riches, Chelsea M. Magin
bioRxiv 2022.05.24.493246; doi: https://doi.org/10.1101/2022.05.24.493246
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