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Gene amplification acts as a molecular foothold to facilitate cross-species adaptation and evasion of multiple antiviral pathways
Shefali Banerjee, View ORCID ProfileCathy Smith, View ORCID ProfileAdam Geballe, View ORCID ProfileStefan Rothenburg, View ORCID ProfileJacob O. Kitzman, View ORCID ProfileGreg Brennan
doi: https://doi.org/10.1101/2022.06.06.494757
Shefali Banerjee
1Department of Biochemistry and Molecular Biology, University of Texas Medical Branch Galveston, Texas, USA
Cathy Smith
2Departments of Human Genetics and Computational Medicine & Bioinformatics, University of Michigan, Ann Arbor, MI 48109, USA
Adam Geballe
3Divisions of Human Biology and Clinical Research, Fred Hutchinson Cancer Center, Seattle, WA, 98109 USA
4Departments of Microbiology and Medicine, University of Washington, Seattle, WA, 98195, USA
Stefan Rothenburg
5Department of Medical Microbiology and Immunology, School of Medicine, University of California, Davis, Davis, CA 95616, USA
Jacob O. Kitzman
2Departments of Human Genetics and Computational Medicine & Bioinformatics, University of Michigan, Ann Arbor, MI 48109, USA
Greg Brennan
5Department of Medical Microbiology and Immunology, School of Medicine, University of California, Davis, Davis, CA 95616, USA
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Posted July 05, 2022.
Gene amplification acts as a molecular foothold to facilitate cross-species adaptation and evasion of multiple antiviral pathways
Shefali Banerjee, Cathy Smith, Adam Geballe, Stefan Rothenburg, Jacob O. Kitzman, Greg Brennan
bioRxiv 2022.06.06.494757; doi: https://doi.org/10.1101/2022.06.06.494757
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