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Prenatal arsenic exposure stymies gut butyrate production and enhances gut permeability in post natal life even in absence of arsenic deftly through miR122-Occludin pathway
View ORCID ProfileMainak Chakraborty, Anupam Gautam, Oishika Das, Aaheli Masid, Moumita Bhaumik
doi: https://doi.org/10.1101/2022.06.13.496028
Mainak Chakraborty
1ICMR-National Institute of Cholera and Enteric Diseases, Beleghata, Kolkata 700010, India
Anupam Gautam
2Institute for Bioinformatics and Medical Informatics, University of Tübingen, Sand 14, 72076 Tübingen, Germany
3International Max Planck Research School “From Molecules to Organisms’’, Max Planck Institute for Biology Tübingen, Max-Planck-Ring∼5, 72076 Tübingen, Germany
Oishika Das
1ICMR-National Institute of Cholera and Enteric Diseases, Beleghata, Kolkata 700010, India
Aaheli Masid
1ICMR-National Institute of Cholera and Enteric Diseases, Beleghata, Kolkata 700010, India
Moumita Bhaumik
1ICMR-National Institute of Cholera and Enteric Diseases, Beleghata, Kolkata 700010, India
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Posted June 16, 2022.
Prenatal arsenic exposure stymies gut butyrate production and enhances gut permeability in post natal life even in absence of arsenic deftly through miR122-Occludin pathway
Mainak Chakraborty, Anupam Gautam, Oishika Das, Aaheli Masid, Moumita Bhaumik
bioRxiv 2022.06.13.496028; doi: https://doi.org/10.1101/2022.06.13.496028
Prenatal arsenic exposure stymies gut butyrate production and enhances gut permeability in post natal life even in absence of arsenic deftly through miR122-Occludin pathway
Mainak Chakraborty, Anupam Gautam, Oishika Das, Aaheli Masid, Moumita Bhaumik
bioRxiv 2022.06.13.496028; doi: https://doi.org/10.1101/2022.06.13.496028
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