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Human liver organoids model progressive inflammatory and fibrotic injury in non-alcoholic fatty liver disease
View ORCID ProfileAnja Hess, Stefan D. Gentile, Amel Ben Saad, Raza-Ur Rahman, Tim Habboub, Alan C. Mullen
doi: https://doi.org/10.1101/2022.07.19.500693
Anja Hess
1Division of Gastroenterology, Massachusetts General Hospital, Boston, MA, USA
2Max Planck Institute for Molecular Genetics, Department of Genome Regulation, Berlin, Germany
Stefan D. Gentile
1Division of Gastroenterology, Massachusetts General Hospital, Boston, MA, USA
3Harvard Medical School, Boston, MA, USA
Amel Ben Saad
1Division of Gastroenterology, Massachusetts General Hospital, Boston, MA, USA
3Harvard Medical School, Boston, MA, USA
Raza-Ur Rahman
1Division of Gastroenterology, Massachusetts General Hospital, Boston, MA, USA
3Harvard Medical School, Boston, MA, USA
Tim Habboub
1Division of Gastroenterology, Massachusetts General Hospital, Boston, MA, USA
Alan C. Mullen
1Division of Gastroenterology, Massachusetts General Hospital, Boston, MA, USA
3Harvard Medical School, Boston, MA, USA
4Harvard Stem Cell Institute, Cambridge, MA, USA
5Klarman Cell Observatory, Broad Institute of MIT and Harvard, Cambridge, MA, USA
6Center for the Study of Inflammatory Bowel Disease, Massachusetts General Hospital, Boston, MA, USA

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Posted July 20, 2022.
Human liver organoids model progressive inflammatory and fibrotic injury in non-alcoholic fatty liver disease
Anja Hess, Stefan D. Gentile, Amel Ben Saad, Raza-Ur Rahman, Tim Habboub, Alan C. Mullen
bioRxiv 2022.07.19.500693; doi: https://doi.org/10.1101/2022.07.19.500693
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