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Chitinase-like proteins promoting tumorigenesis through disruption of cell polarity via enlarged endosomal vesicles

Dilan Khalili, Martin Kunc, Sarah Herbrich, Anna Mueller, Ulrich Theopold
doi: https://doi.org/10.1101/2022.08.17.504232
Dilan Khalili
1The Wenner-Gren Institute, Department of Molecular Biosciences, Stockholm University, Stockholm, Sweden
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Martin Kunc
1The Wenner-Gren Institute, Department of Molecular Biosciences, Stockholm University, Stockholm, Sweden
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Sarah Herbrich
1The Wenner-Gren Institute, Department of Molecular Biosciences, Stockholm University, Stockholm, Sweden
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Anna Mueller
1The Wenner-Gren Institute, Department of Molecular Biosciences, Stockholm University, Stockholm, Sweden
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Ulrich Theopold
1The Wenner-Gren Institute, Department of Molecular Biosciences, Stockholm University, Stockholm, Sweden
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  • For correspondence: uli.theopold@su.se
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Article Information

doi 
https://doi.org/10.1101/2022.08.17.504232
History 
  • January 24, 2023.

Article Versions

  • Version 1 (August 17, 2022 - 16:34).
  • You are currently viewing Version 2 of this article (January 24, 2023 - 03:12).
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Copyright 
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.

Author Information

  1. Dilan Khalili1,
  2. Martin Kunc1,
  3. Sarah Herbrich1,
  4. Anna Mueller1 and
  5. Ulrich Theopold1,*
  1. 1The Wenner-Gren Institute, Department of Molecular Biosciences, Stockholm University, Stockholm, Sweden
  1. ↵*Correspondence:
    Ulrich Theopold, uli.theopold{at}su.se
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Posted January 24, 2023.
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Chitinase-like proteins promoting tumorigenesis through disruption of cell polarity via enlarged endosomal vesicles
Dilan Khalili, Martin Kunc, Sarah Herbrich, Anna Mueller, Ulrich Theopold
bioRxiv 2022.08.17.504232; doi: https://doi.org/10.1101/2022.08.17.504232
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Chitinase-like proteins promoting tumorigenesis through disruption of cell polarity via enlarged endosomal vesicles
Dilan Khalili, Martin Kunc, Sarah Herbrich, Anna Mueller, Ulrich Theopold
bioRxiv 2022.08.17.504232; doi: https://doi.org/10.1101/2022.08.17.504232

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