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DPYSL2/CRMP2 isoform B knockout in human iPSC-derived glutamatergic neurons confirms its role in mTOR signaling and neurodevelopmental disorders
Kyra L. Feuer, Xi Peng, Christian Yovo, Dimitri Avramopoulos
doi: https://doi.org/10.1101/2022.11.19.517191
Kyra L. Feuer
1Department of Genetic Medicine, Johns Hopkins University School of Medicine
2Department of Biomedical Engineering, Whiting School of Engineering and School of Medicine, Johns Hopkins University
Xi Peng
1Department of Genetic Medicine, Johns Hopkins University School of Medicine
Christian Yovo
1Department of Genetic Medicine, Johns Hopkins University School of Medicine
Dimitri Avramopoulos
1Department of Genetic Medicine, Johns Hopkins University School of Medicine
3Department of Psychiatry, Johns Hopkins University School of Medicine
Posted November 19, 2022.
DPYSL2/CRMP2 isoform B knockout in human iPSC-derived glutamatergic neurons confirms its role in mTOR signaling and neurodevelopmental disorders
Kyra L. Feuer, Xi Peng, Christian Yovo, Dimitri Avramopoulos
bioRxiv 2022.11.19.517191; doi: https://doi.org/10.1101/2022.11.19.517191
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