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Fetal hematopoietic stem cells are activated during acute prenatal infection with Toxoplasma gondii and IFNγ
View ORCID ProfileApril C. Apostol, View ORCID ProfileKelly S. Otsuka, View ORCID ProfileDiego A. López, View ORCID ProfileJasmine Posada, View ORCID ProfileKirk D.C. Jensen, View ORCID ProfileAnna E. Beaudin
doi: https://doi.org/10.1101/2022.11.29.518417
April C. Apostol
1Quantitative and Systems Biology Graduate Program, University of California-Merced, Merced, CA, USA
Kelly S. Otsuka
2Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT, USA
Diego A. López
2Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT, USA
Jasmine Posada
3Department of Molecular and Cell Biology, University of California, Merced, Merced, CA, USA
Kirk D.C. Jensen
3Department of Molecular and Cell Biology, University of California, Merced, Merced, CA, USA
4Health Science Research Institute, University of California, Merced, Merced, CA, USA
Anna E. Beaudin
5Departments of Internal Medicine and Pathology, and Program in Molecular Medicine, University of Utah School of Medicine, Salt Lake City, UT, USA

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Posted November 29, 2022.
Fetal hematopoietic stem cells are activated during acute prenatal infection with Toxoplasma gondii and IFNγ
April C. Apostol, Kelly S. Otsuka, Diego A. López, Jasmine Posada, Kirk D.C. Jensen, Anna E. Beaudin
bioRxiv 2022.11.29.518417; doi: https://doi.org/10.1101/2022.11.29.518417
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