Article Information
History
- December 2, 2022.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Jian Fenga,b,
- Wen Menga,b,
- Luping Chena,b,
- Xinquan Zhanga,b,
- Ashley Markazia,b,
- Weiming Yuanc,
- Yufei Huanga,d,e,f and
- Shou-Jiang Gaoa,b,*
- aCancer Virology Program, UPMC Hillman Cancer Center, University of Pittsburgh, Pittsburgh, PA, USA
- bDepartment of Microbiology and Molecular Genetics, University of Pittsburgh, Pittsburgh, PA, USA
- cDepartment of Molecular Microbiology and Immunology, Keck School of Medicine, University of Southern California School of Medicine, Los Angeles, CA, USA
- dDepartment of Electrical and Computer Engineering, University of Pittsburgh, Pittsburgh, PA, USA
- eDepartment of Biomedical Informatics, University of Pittsburgh, Pittsburgh, PA, USA
- fDepartment of Medicine, University of Pittsburgh, Pittsburgh, PA, USA
- ↵*Address correspondence to Shou-Jiang Gao, gaos8{at}upmc.edu.
Posted December 02, 2022.
N6-methyladenosine (m6A) and reader protein YTHDF2 enhance innate immune response by mediating DUSP1 mRNA degradation and activating mitogen-activated protein kinases during bacterial and viral infections
