Article Information
History
- December 23, 2022.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Shugo Sasaki1,2,
- Cuilan Nian1,2,3,
- Eric E. Xu1,3,
- Daniel J. Pasula1,2,
- Helena Winata1,2,3,
- Sanya Grover1,
- Dan S. Luciani1,2 and
- Francis C. Lynn1,2,3,4,#
- 1BC Children’s Hospital Research Institute, Vancouver, BC
- 2Department of Surgery, The University of British Columbia, Vancouver, BC
- 3Department of Cellular and Physiological Sciences, The University of British Columbia, Vancouver, BC
- 4School of Biomedical Engineering, The University of British Columbia, Vancouver, BC
- ↵#Corresponding author: Francis C. Lynn, Department of Surgery, Department of Cellular & Physiological Sciences, The University of British Columbia BC Children’s Hospital Diabetes Research Group, 950 28th Ave W., Vancouver, BC Canada V5Z 4H4, Tel: 604 875 2000 x5426, Fax: 604 875 2373, E-mail: francis.lynn{at}ubc.ca, Twitter: @nictitate; @beta_cell
Posted December 23, 2022.
Type 2 diabetes susceptibility gene GRK5 regulates physiological pancreatic β-cell proliferation via phosphorylation of HDAC5 in mice
