New Results
Type 2 diabetes susceptibility gene GRK5 regulates physiological pancreatic β-cell proliferation via phosphorylation of HDAC5 in mice
View ORCID ProfileShugo Sasaki, Cuilan Nian, View ORCID ProfileEric E. Xu, View ORCID ProfileDaniel J. Pasula, View ORCID ProfileHelena Winata, Sanya Grover, View ORCID ProfileDan S. Luciani, View ORCID ProfileFrancis C. Lynn
doi: https://doi.org/10.1101/2022.12.23.521810
Shugo Sasaki
1BC Children’s Hospital Research Institute, Vancouver, BC
2Department of Surgery, The University of British Columbia, Vancouver, BC
Cuilan Nian
1BC Children’s Hospital Research Institute, Vancouver, BC
2Department of Surgery, The University of British Columbia, Vancouver, BC
3Department of Cellular and Physiological Sciences, The University of British Columbia, Vancouver, BC
Eric E. Xu
1BC Children’s Hospital Research Institute, Vancouver, BC
3Department of Cellular and Physiological Sciences, The University of British Columbia, Vancouver, BC
Daniel J. Pasula
1BC Children’s Hospital Research Institute, Vancouver, BC
2Department of Surgery, The University of British Columbia, Vancouver, BC
Helena Winata
1BC Children’s Hospital Research Institute, Vancouver, BC
2Department of Surgery, The University of British Columbia, Vancouver, BC
3Department of Cellular and Physiological Sciences, The University of British Columbia, Vancouver, BC
Sanya Grover
1BC Children’s Hospital Research Institute, Vancouver, BC
Dan S. Luciani
1BC Children’s Hospital Research Institute, Vancouver, BC
2Department of Surgery, The University of British Columbia, Vancouver, BC
Francis C. Lynn
1BC Children’s Hospital Research Institute, Vancouver, BC
2Department of Surgery, The University of British Columbia, Vancouver, BC
3Department of Cellular and Physiological Sciences, The University of British Columbia, Vancouver, BC
4School of Biomedical Engineering, The University of British Columbia, Vancouver, BC
Article usage
Posted December 23, 2022.
Type 2 diabetes susceptibility gene GRK5 regulates physiological pancreatic β-cell proliferation via phosphorylation of HDAC5 in mice
Shugo Sasaki, Cuilan Nian, Eric E. Xu, Daniel J. Pasula, Helena Winata, Sanya Grover, Dan S. Luciani, Francis C. Lynn
bioRxiv 2022.12.23.521810; doi: https://doi.org/10.1101/2022.12.23.521810
Type 2 diabetes susceptibility gene GRK5 regulates physiological pancreatic β-cell proliferation via phosphorylation of HDAC5 in mice
Shugo Sasaki, Cuilan Nian, Eric E. Xu, Daniel J. Pasula, Helena Winata, Sanya Grover, Dan S. Luciani, Francis C. Lynn
bioRxiv 2022.12.23.521810; doi: https://doi.org/10.1101/2022.12.23.521810
Subject Area
Subject Areas
- Biochemistry (13697)
- Bioengineering (10429)
- Bioinformatics (33141)
- Biophysics (17097)
- Cancer Biology (14169)
- Cell Biology (20098)
- Clinical Trials (138)
- Developmental Biology (10866)
- Ecology (16008)
- Epidemiology (2067)
- Evolutionary Biology (20334)
- Genetics (13392)
- Genomics (18629)
- Immunology (13741)
- Microbiology (32149)
- Molecular Biology (13380)
- Neuroscience (70030)
- Paleontology (526)
- Pathology (2188)
- Pharmacology and Toxicology (3741)
- Physiology (5860)
- Plant Biology (12020)
- Synthetic Biology (3365)
- Systems Biology (8163)
- Zoology (1841)