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- September 17, 2024.
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- Version 1 (August 16, 2023 - 08:52).
- You are viewing Version 2, the most recent version of this article.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC 4.0 International license.
Author Information
- Benjamin Boumard1,2,
- Gwenn Le Meur1,
- Marine Stefanutti1,
- Tania Maalouf1,
- Marwa El-Hajj1,
- Reinhard Bauer3 and
- Allison J. Bardin1,4,#
- 1Institut Curie, PSL Research University, Sorbonne University, CNRS UMR 3215, INSERM U934, Stem Cells and Tissue Homeostasis Group, Paris, France
- 2Institute of Cellular and Organismic Biology (ICOB), Academia Sinica, Taipei, Taiwan
- 3LIMES-Institute, Development, Genetics and Mol. Physiology Unit, Dpt. For Molecular Developmental Biology, University of Bonn, Carl-Troll-Str. 31, D-53115 Bonn, Germany
- ↵#Corresponding author. Email: allison.bardin{at}curie.fr
Posted September 17, 2024.
Cell-type-specific nucleotide sharing through gap junctions impacts sensitivity to replication stress
