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- July 31, 2024.
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- Version 1 (January 26, 2024 - 13:24).
- You are viewing Version 2, the most recent version of this article.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY 4.0 International license.
Author Information
- Eleni C.R. Hackwell1,2,
- Sharon R. Ladyman1,2,4,
- Jenny Clarkson1,3,
- H. James McQuillan1,2,
- Ulrich Boehm5,
- Allan E. Herbison6,
- Rosemary S.E. Brown1,3 and
- David R. Grattan1,2,4,*
- 1Centre for Neuroendocrinology, School of Biomedical Sciences, University of Otago, Dunedin, NZ
- 2Department of Anatomy, School of Biomedical Sciences, University of Otago, Dunedin, NZ
- 3Department of Physiology, School of Biomedical Sciences, University of Otago, Dunedin, NZ
- 4Maurice Wilkins Centre for Molecular Biodiscovery, Auckland, NZ
- 5Saarland University School of Medicine, Centre for Molecular Signalling (PZMS), Experimental Pharmacology, Homburg, Germany
- 6Department of Physiology, Development and Neuroscience, University of Cambridge, Cambridge, United Kingdom
- ↵*Corresponding author: David R. Grattan Email:
dave.grattan{at}otago.ac.nz
Posted July 31, 2024.
Prolactin-mediates a lactation-induced suppression of arcuate kisspeptin neuronal activity necessary for lactational infertility in mice
