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- November 11, 2024.
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- Version 1 (April 1, 2024 - 10:25).
- You are viewing Version 2, the most recent version of this article.
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The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Alex Orlowski1,
- Joseph Karippaparambil1,
- Jean-Michel Paumier1,#,
- Shraddha Ghanta1,#,
- Eduardo Pallares1,
- Rumamol Chandran1,
- Daisy Edmison1,
- Jamuna Tandukar2,
- Ruixuan Gao2,3 and
- Swetha Gowrishankar1,*
- 1Department of Anatomy and Cell Biology, College of Medicine, University of Illinois Chicago, Chicago, IL 60612 USA
- 2Department of Biological Sciences, College of Liberal Arts and Sciences, University of Illinois Chicago, Chicago, IL 60607 USA
- 3Department of Chemistry, College of Liberal Arts and Sciences, University of Illinois Chicago, Chicago, IL 60607 USA
- ↵*Correspondence to: Swetha Gowrishankar (swethag{at}uic.edu)
↵# These authors contributed equally to the work
Posted November 11, 2024.
Axonal organelle buildup from loss of AP-4 complex function causes exacerbation of amyloid plaque pathology and gliosis in Alzheimer’s disease mouse model
