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Steady-state, therapeutic, and helminth-induced IL-4 compromise protective CD8 T cell bystander activation
View ORCID ProfileNicholas J Maurice, Talia S Dalzell, View ORCID ProfileNicholas N Jarjour, Taylor A DePauw, Stephen C Jameson
doi: https://doi.org/10.1101/2024.06.10.598293
Nicholas J Maurice
1Center for Immunology, University of Minnesota Medical School, Minneapolis, MN
2Department of Laboratory Medicine and Pathology, University of Minnesota Medical School, Minneapolis, MN
Talia S Dalzell
1Center for Immunology, University of Minnesota Medical School, Minneapolis, MN
2Department of Laboratory Medicine and Pathology, University of Minnesota Medical School, Minneapolis, MN
Nicholas N Jarjour
1Center for Immunology, University of Minnesota Medical School, Minneapolis, MN
2Department of Laboratory Medicine and Pathology, University of Minnesota Medical School, Minneapolis, MN
Taylor A DePauw
1Center for Immunology, University of Minnesota Medical School, Minneapolis, MN
2Department of Laboratory Medicine and Pathology, University of Minnesota Medical School, Minneapolis, MN
Stephen C Jameson
1Center for Immunology, University of Minnesota Medical School, Minneapolis, MN
2Department of Laboratory Medicine and Pathology, University of Minnesota Medical School, Minneapolis, MN
Posted June 12, 2024.
Steady-state, therapeutic, and helminth-induced IL-4 compromise protective CD8 T cell bystander activation
Nicholas J Maurice, Talia S Dalzell, Nicholas N Jarjour, Taylor A DePauw, Stephen C Jameson
bioRxiv 2024.06.10.598293; doi: https://doi.org/10.1101/2024.06.10.598293
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