Article Information
History
- August 1, 2024.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. This article is a US Government work. It is not subject to copyright under 17 USC 105 and is also made available for use under a CC0 license.
Author Information
- Liam Horan-Portelance1,
- Michiyo Iba1,
- Dominic J. Acri2,
- J. Raphael Gibbs3,
- Eliezer Masliah1,4 and
- Mark R. Cookson2,*
- 1Molecular Neuropathology Unit, Laboratory of Neurogenetics, National Institute on Aging, National Institutes of Health, Bethesda, MD 20892, USA
- 2Cell Biology and Gene Expression Section, Laboratory of Neurogenetics, National Institute on Aging, National Institutes of Health, Bethesda, MD 20892, USA
- 3Computational Biology Group, Laboratory of Neurogenetics, National Institute on Aging, National Institutes of Health, Bethesda, MD 20892, USA
- 4Division of Neuroscience, National Institute on Aging, National Institutes of Health, Bethesda, MD 20892, USA
- ↵*Corresponding author: Mark R. Cookson, PhD, Laboratory of Neurogenetics, NIA, NIH, Building 35, Room 1A116, MSC3707 35, Convent Drive, Bethesda, MD, 20892-3707, USA. Phone +1-301-451-3870. Email: cookson{at}mail.nih.gov
Posted August 01, 2024.
Single-cell spatial transcriptomics reveals molecular patterns of selective neuronal vulnerability to α-synuclein pathology in a transgenic mouse model of Lewy body disease
